, 2001). This constrained malignant development relates prominently on the activation of p53 in aspiring cancer cells encountering telomere dysfunction. Inactivation of the p53 tumor suppressor is Probably the most Recurrent functions in human epithelial cancers (Brosh and Rotter, 2009) and the standing of p53 can dictate regardless of whether https://janel246csj7.ltfblog.com/profile